Olympic Health

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Benefits of Health in Olympic Circles

The Benefits of Olympic Health

Ask any Olympic Athlete

You will hear that a healthy mind and body are extremely important variables in the odds of winning any competition, let alone Olympic competition. At Body and Soil Nutrition we offer a program we call “Olympic Health” which is designed specifically with individuality in mind. No two competitors have the same health needs and neither do you. So why would you follow a health plan designed for someone else? I don’t know either.

Consider Individuality in Health

It’s easy to see that individuality is so prevalent in the world of natural health. 1000’s of books have been written by a multitude of authors and they all have one thing in common. They only work for a certain type of person. They don’t work for everybody. Why do you think that is?

Beyond blood type, beyond metabolic type, beyond Pitta-Kapha-Vata, beyond the 5 Chinese elements; we have body chemistry. There are no two people on the face of this earth with exactly the same chemistry make-up. Just like no two people on this earth with exactly the same DNA. By sequencing the DNA from various people, scientists have found 15 million single nucleotide polymorphisms, 1 million short insertions and deletions, and 20,000 structural variants (The 1000 Genomes Project Consortium). Assuming each of these sites of variation has only two alleles and that none are synthetically lethal, this puts an estimate of the possible combinations from randomly assorting these variants at 2^(1.6×10^6) or 10^480000.
Read more: http://www.physicsforums.com

Take Your Own Chemistry & Formulate Your Own Healthy Living Plan

In our Olympic Health program, we teach you how to use a fresh sample of your own urine and saliva to formulate a health plan based on your own uniqueness. What is right for you to eat probably won’t be right for a brother or sister, mother or father. Because of the differences in DNA, there are differences in the individual dietary and mineralization needs of each and every one of you.  You can’t eat like anyone else and expect to get the same results as them. It just doesn’t work that way. Now, if you eat the typical Twinkie and coke diet, something is going to break down in your body. You know you’re going to end up sick someday, maybe you just hope it won’t be soon. But you know, without question, that trying to live off that junk is going to kill you. What you don’t know is which system of your body is going to break down first. Maybe you’ll end up with heart disease, maybe Diabetes or even cancer. Without the mineral energy necessary for proper functioning of the body and water to take out the garbage, whatever part of your body is genetically weakest is going to succumb first to disease.

Wealth, Prestige, Confidence, Love and Time Are Yours for the Taking

Olympic Health and the benefits are yours for the taking. If you live in the United States, you have access to the training and the equipment necessary to take control of your own health; Olympic Health! So, why don’t you take control of your own health? What is stopping you from changing your life?

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A quick interview about GMO, Glyphosate, RoundUp….And the issues they can cause.

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GMOs and Diseases Exclusive Interview with Jeffrey Smith by Dr. Pompa on Genetically Modified Organisms.
New information is always being uncovered about the dangers of GMO foods and the RoundUp they were designed for. Not only is it obvious that RoundUp and Glyphosate (the main herbicide in RoundUp) are involved in GMO foods, but you will also find grains full of glyphosate. I know it’s more expensive to buy 100% organic, but you can’t avoid Monsanto’s products unless you do. Which is more expensive, health or sickness? In order to maintain health we have to band together and start using only 100% organic or herbicide-free food!

WSU Veterinary College warns animal owners of tick paralysis

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March 19, 2014

By Charlie Powell, College of Veterinary Medicine

Rocky Mountain wood tick

PULLMAN, Wash. – Warming weather in the Pacific Northwest brings with it a renewed threat of tick paralysis in animals and people.

Tick paralysis is a somewhat uncommon but potentially fatal disease that can affect virtually all warm-blooded land animals. The illness occurs when certain species of ticks inject potent toxins from their salivary glands into the host animal.

The disease was first identified in Australia in 1824. Since then, more than 60 species of ticks worldwide have been identified as toxin producers.

At least two prominent species of ticks on the list of toxin producers are native to North America, including the Pacific Northwest.  In fact, in North America, ticks actually transmit more disease than mosquitoes. Tick paralysis occurs most in the northwestern and southeastern regions of North America along with the east coast of Australia and southern Africa.

“Both human and veterinary medical professionals know tick paralysis can be a serious, potentially fatal illness,” explained Steve Parish, a large animal veterinarian who heads up Washington State University’s Agricultural Animal Service. “Llamas and alpacas seem to be particularly susceptible to the effects of the toxin and we don’t know why.”

Each spring and summer, WSU’s veterinary teaching hospital sees or provides consultation on several cases of tick paralysis in llamas and alpacas.

The cases come from throughout the Northwest. Occurrence of the disease is sporadic and is difficult to predict because the toxin does not occur in all tick populations all of the time or in the same local regions. And only female ticks attach firmly and engorge on blood from the hosts.

“Symptoms of tick paralysis include a rapidly progressing total paralysis over 24 to 72 hours after the ticks attach themselves to the animal,” said Parish. “Untreated, an animal may be unable to chew, swallow, drink or breathe as the paralysis progresses to the respiratory system. Obviously, if the breathing mechanism is paralyzed, the animal dies.”

Death rates for tick paralysis have been reported as high as 10 percent or more in humans and around 7 percent in domestic animals. Medical literature shows that in most human cases of the disease, only a single tick was found on the patient.

“As dire as this disease sounds, though, treatment is effective and reversal of the paralysis can be rapid with an excellent prognosis,” said Parish. “The key is to remove the ticks and provide supportive care.

“Recovery occurs very quickly in most animals and people within 48 hours, but llamas and alpacas seem to be hit a harder than other animals for reasons not well understood,” he said. “Complete recovery could take up to several weeks in those species.”

Removing all ticks may require that an animal be shorn in addition to administering medication that kills any ticks still attached.

“Llama and alpaca owners working with their veterinarian will find the drug ivermectin to be very effective when used in the proper dosage,” said Parish.  “In addition to tick removal and ivermectin, it may also be necessary to use external chemical insecticides such as the pyrethrins. Certainly, tick repellants can help prevent ticks from attaching.”

“Ticks are best removed with curved tweezers, forceps or gloved fingers,” said Parish. The tick should be grasped close to the skin and pulled with steady but gentle traction to ensure all the mouth parts come off with the entire tick.

Other methods of tick removal famous as folklore, such as applying a lighted match or petroleum jelly to the tick, are not effective.

“After removing a tick, people should always wash their hands thoroughly with soap and water,” Parish said.



Charlie Powell, WSU College of Veterinary Medicine communications, 509-335-7073,cpowell@vetmed.wsu.edu

146 Reasons Sugar is Ruining Your Health

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146 Reasons Why Sugar Is Ruining Your Health
Nancy Appleton, Ph.D.

I really enjoyed Nancy’s List of the effects sugar has on the body. Please read and enjoy!!

1.  Sugar can suppress the immune system.
2.  Sugar upsets the mineral relationships in the body.
3.  Sugar can cause hyperactivity, anxiety, difficulty concentrating, and crankiness in children.
4.  Sugar can produce a significant rise in triglycerides.
5.  Sugar contributes to the reduction in defense against bacterial infection (infectious diseases).
6.  Sugar causes a loss of tissue elasticity and function, the more sugar you eat the more elasticity     and function you lose.
7.  Sugar reduces high-density lipoproteins.
8.  Sugar leads to chromium deficiency.
9.  Sugar leads to cancer of the ovaries.
10. Sugar can increase fasting levels of glucose.
11. Sugar causes copper deficiency.
12. Sugar interferes with absorption of calcium and magnesium.
13. Sugar may make eyes more vulnerable to age-related macular degeneration.
14. Sugar raises the level of neurotransmitters: dopamine, serotonin, and norepinephrine.
15. Sugar can cause hypoglycemia.
16. Sugar can produce an acidic digestive tract.
17. Sugar can cause a rapid rise of adrenaline levels in children.
18. Sugar malabsorption is frequent in patients with functional bowel disease.
19. Sugar can cause premature aging.
20. Sugar can lead to alcoholism.
21. Sugar can cause tooth decay.
22. Sugar contributes to obesity
23. High intake of sugar increases the risk of Crohn’s disease, and ulcerative colitis.
24. Sugar can cause changes frequently found in person with gastric or duodenal ulcers.
25. Sugar can cause arthritis.
26. Sugar can cause asthma.
27. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).
28. Sugar can cause gallstones.
29. Sugar can cause heart disease.
30. Sugar can cause appendicitis.
31. Sugar can cause hemorrhoids.
32. Sugar can cause varicose veins.
33. Sugar can elevate glucose and insulin responses in oral contraceptive users.
34. Sugar can lead to periodontal disease.
35. Sugar can contribute to osteoporosis.
36. Sugar contributes to saliva acidity.
37. Sugar can cause a decrease in insulin sensitivity.
38. Sugar can lower the amount of Vitamin E (alpha-Tocopherol) in the blood.
39. Sugar can decrease growth hormone.
40. Sugar can increase cholesterol.
41. Sugar can increase the systolic blood pressure.
42. High sugar intake increases advanced glycation end products (AGEs)(Sugar bound non-enzymatically to protein)
43. Sugar can interfere with the absorption of protein.
44. Sugar causes food allergies.
45. Sugar can contribute to diabetes.
46. Sugar can cause toxemia during pregnancy.
47. Sugar can contribute to eczema in children.
48. Sugar can cause cardiovascular disease.
49. Sugar can impair the structure of DNA
50. Sugar can change the structure of protein.
51. Sugar can make our skin age by changing the structure of collagen.
52. Sugar can cause cataracts.
53. Sugar can cause emphysema.
54. Sugar can cause atherosclerosis.
55. Sugar can promote an elevation of low-density lipoproteins (LDL).
56. High sugar intake can impair the physiological homeostasis of many systems in the body.
57. Sugar lowers the enzymes ability to function.
58. Sugar intake is higher in people with Parkinson’s disease.
59. Sugar can increase the size of the liver by making the liver cells divide.
60. Sugar can increase the amount of liver fat.
61. Sugar can increase kidney size and produce pathological changes in the kidney.
62. Sugar can damage the pancreas.
63. Sugar can increase the body’s fluid retention.
64. Sugar is enemy #1 of the bowel movement.
65. Sugar can cause myopia (nearsightedness).
66. Sugar can compromise the lining of the capillaries.
67. Sugar can make the tendons more brittle.
68. Sugar can cause headaches, including migraine.
69. Sugar plays a role in pancreatic cancer in women.
70. Sugar can adversely affect school children’s grades and cause learning disorders.
71. Sugar can cause depression.
72. Sugar increases the risk of gastric cancer.
73. Sugar and cause dyspepsia (indigestion).
74. Sugar can increase your risk of getting gout.
75. Sugar can increase the levels of glucose in an oral glucose tolerance test over the ingestion of complex carbohydrates.
76. Sugar can increase the insulin responses in humans consuming high-sugar diets compared to low-sugar diets.
77. A diet high in refined sugar reduces learning capacity.
78. Sugar can cause less effective functioning of two blood proteins, albumin, and lipoproteins, which may reduce the body’s ability to handle fat and cholesterol.
79.  Sugar can contribute to Alzheimer’s disease.
80. Sugar can cause platelet adhesiveness.
81. Sugar can cause hormonal imbalance; some hormones become under active and others become overactive.
82. Sugar can lead to the formation of kidney stones.
83. Diets high in sugar can cause free radicals and oxidative stress.
84. High sugar diet can lead to biliary tract cancer.
85. High sugar consumption of pregnant adolescents is associated with a twofold-increased risk for delivering a small-for-gestational-age (SGA) infant.
86. High sugar consumption can lead to substantial decrease in gestation duration among adolescents.
87. Sugar slows food’s travel time through the gastrointestinal tract.
88. Sugar increases the concentration of bile acids in stools and bacterial enzymes in the colon. This can modify bile to produce cancer-causing compounds and colon cancer.
89.  Sugar increases estradiol (the most potent form of naturally occurring estrogen) in men.
90.  Sugar combines with and destroys phosphatase, an enzyme, which makes the process of digestion more difficult.
91.  Sugar can be a risk factor of gallbladder cancer.
92. Sugar is an addictive substance.
93. Sugar can be intoxicating, similar to alcohol.
94. Sugar can exacerbate PMS.
95. Sugar given to premature babies can affect the amount of carbon dioxide they produce.
96. Decrease in sugar intake can increase emotional stability.
97. The rapid absorption of sugar promotes excessive food intake in obese subjects.
98. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).
99. Sugar adversely affects urinary electrolyte composition.
100. Sugar can slow down the ability of the adrenal glands to function.
101. I.Vs (intravenous feedings) of sugar water can cut off oxygen to the brain.
102. High sucrose intake could be an important risk factor in lung cancer.
103. Sugar increases the risk of polio.
104. High sugar intake can cause epileptic seizures.
105. Sugar causes high blood pressure in obese people.
106. In Intensive Care Units, limiting sugar saves lives.
107. Sugar may induce cell death.
108. Sugar can increase the amount of food that you eat.
109. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44% drop in antisocial behavior.
110.  Sugar can lead to prostate cancer.
111. Sugar dehydrates newborns.
112.  Sugar can cause low birth weight babies.
113. Greater consumption of refined sugar is associated with a worse outcome of schizophrenia
114. Sugar can raise homocysteine levels in the blood stream.
115. Sweet food items increase the risk of breast cancer.
116. Sugar is a risk factor in cancer of the small intestine.
117. Sugar may cause laryngeal cancer.
118. Sugar induces salt and water retention.
119. Sugar may contribute to mild memory loss.
120. The more sodas a 10 year old child consumes, the less milk.
121. Sugar can increase the total amount of food consumed.
122. Exposing a newborn to sugar results in a heightened preference for sucrose relative to water at 6 months and 2 years of age.
123.  Sugar causes constipation.
124.  Sugar causes varicose veins.
125.  Sugar can cause brain decay in prediabetic and diabetic women.
126.  Sugar can increase the risk of stomach cancer.
127.  Sugar can cause metabolic syndrome.
128.  Sugar ingestion by pregnant women increases neural tube defects in embryos.
129.  Sugar can be a factor in asthma.
130.  The higher the sugar consumption the more chances of getting irritable bowel syndrome.
131.  Sugar can affect the brain’s ability to deal with rewards and consequences.
132.  Sugar can cause cancer of the rectum.
133.  Sugar can cause endometrial cancer.
134.  Sugar can cause renal (kidney) cell carcinoma.
135.  Sugar can cause liver tumors.
136.   Sugar can increase inflammatory markers in the blood stream of overweight people.
137. Sugar can lower Vitamin E levels in the blood stream.
138. Sugar can increase your appetite for all food.
139. Sugar plays a role in the etiology and the continuation of acne.
140. Too much sugar can kill your sex life.
141. Sugar saps school performance in children.
142. Sugar can cause fatigue, moodiness, nervousness and depression.
143.   Sugar is common choice of obese individuals.
144.   A linear decrease in the intake of many essential nutrients is associated with increasing total sugar intake.
145. High fructose consumption has been linked to liver disease.
146. Sugar adds to the risk of bladder cancer.

1. Sanchez, A., et al. “Role of Sugars in Human Neutrophilic Phagocytosis,”  Am J Clin Nutr. Nov 1973;261:1180-1184.
Bernstein, J., et al. “Depression of Lymphocyte Transformation Following Oral Glucose Ingestion.” Am J  Clin Nutr. 1997;30:613.
2. Couzy, F., et al.”Nutritional Implications of the Interaction Minerals,” Progressive Food and Nutrition Science. 17;1933:65-87.
3. Goldman, J., et al.  “Behavioral Effects of Sucrose on Preschool Children.” J Abnormal Child Psychol. 1986;14(4):565-577.
4. Scanto, S. and Yudkin, J. “The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers.”  J Postgrad Med. 1969;45:602-607.
5.  Ringsdorf, W., Cheraskin, E. and Ramsay R. “Sucrose,Neutrophilic Phagocytosis and Resistance to Disease,” Dental Surv. 1976;52(12):46-48.
6.  Cerami, A., et al. “Glucose and Aging.” Scientific American. May 1987:90.
Lee, A. T. and Cerami, A. “The Role of Glycation in Aging.” Ann N Y Acad Sci. 663:63-67.
7.  Albrink, M. and Ullrich I. H. “Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets.” Am J Clin  Nutr. 1986;43:419-428.
Pamplona, R., et al. “Mechanisms of Glycation in Atherogenesis.” Med Hypotheses. Mar 1993;40(3):174-81.
8.  Kozlovsky, A., et al.  “Effects of Diets High in Simple Sugars on Urinary Chromium Losses.”  Metabolism. June 1986;35:515-518.
9.  Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41.
10. Kelsay, J., et al. “Diets High in Glucose or Sucrose and Young Women.”  Am J Clin Nutr. 1974;27:926-936.
Thomas, B. J., et al. “Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose,” Hum Nutr Clin Nutr. 1983; 36C(1):49_51.
11. Fields, M.., et al. “Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets,” Am J Clin Nutr. 1983;113:1335-1345.
12. Lemann, J. “Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium.” Am J Clin Nutr. 1976 ;70:236-245.
13. Chiu, C. Am J Clin Nutr. July 2007;86:180-188
14. “Sugar, White Flour Withdrawal Produces Chemical Response.” The Addiction Letter .Jul 1992:4.
15. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
16. Ibid.
17. Jones, T. W., et al. “Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children.” J Pediatrics. Feb 1995;126:171-7.
18. Ibid.
19. Lee, A. T.and Cerami A. “The Role of Glycation in Aging.” Annals of the New York Academy of Science. 1992;663:63-70.
20. Abrahamson, E. and Peget, A. Body, Mind and Sugar. (New York:Avon,1977.)
21. Glinsmann, W., et al. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” F. D. A. Report of Sugars Task Force. 1986:39.
Makinen K.K.,et al. “A Descriptive Report of the Effects of a 16_month Xylitol Chewing_Gum Programme Subsequent to a 40_Month Sucrose Gum Programme.” Caries Research. 1998; 32(2)107-12.
Riva Touger-Decker and Cor van Loveren, “Sugars and Dental Caries.”
Am. J. Clin.Nutr. Oct 2003; 78:881-892.
22. Keen, H., et al. “Nutrient Intake, Adiposity, and Diabetes.” Brit Med J. 1989; 1: 655-658.
23. Tragnone, A. et al. “Dietary Habits as Risk Factors for Inflammatory Bowel Disease.”  Eur J Gastroenterol Hepatol. Jan 1995;7(1):47-51.
24. Yudkin, J.  Sweet and Dangerous. (New York;Bantam Books:1974), 129.
25. Darlington, L., Ramsey, et al. “Placebo-Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis,” Lancet.  Feb 1986;8475(1):236-238.
26. Powers, L. “Sensitivity: You React to What You Eat.” Los Angeles Times. Feb. 12, 1985.
Cheng, J., et al.  “Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors.” Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
27. Crook, W. J.  The Yeast Connection. (TN:Professional Books, 1984).
28. Heaton, K. “The Sweet Road to Gallstones.” Brit Med J. Apr 14, 1984; 288:1103-1104.
Misciagna, G., et al. Am J Clin Nutr. 1999;69:120-126.
29. Yudkin, J. “Sugar Consumption and Myocardial Infarction.” Lancet. Feb 6, 1971;1(7693):296-297.
Chess DJ, et al. “Deleterious Effects of Sugar and Protective Effects of Starch on Cardiac Remodeling, Contractile Dysfunction, and Mortality in Response to Pressure Overload.” Am J Physiol Heart Circ Physiol. Sept. 2007;293(3):H1853-H1860
30. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
31. op. cit.
32. Cleave, T. and Campbell, G. Diabetes, Coronary Thrombosis and the Saccharine Disease. (Bristol, England, John Wright and Sons, 1960).
33. Behall, K. “Influence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters.” Disease Abstracts International. 1982;431-437.
34. Glinsmann, W., et al. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” F. D. A. Report of Sugars Task Force. 1986;39:36_38.
35. Tjäderhane, L. and Larmas, M. “A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats.” Am J Clin Nutr. 1998:128:1807-1810.
36.  Wilson R.F. and Ashley F.P. “The Effects of Experimental Variations in Dietary Sugar Intake and Oral hygiene on the Biochemical Composition and pH of Free Smooth-surface and Approximal Plaque.” J Dent Res. June 1988;67(6):949-953
37. Beck-Nielsen H., et al. “Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects.” Diabetes. 1978;15:289-296.
38. Mohanty P.  et al. “Glucose Challenge Stimulates Reactive Oxygen Species (ROS) Generation by Leucocytes.” J Clin Endocrin Metabol. Aug 2000; 85(8):2970-2973.
39. Gardner, L. and Reiser, S. “Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol.”  Proc Soc Exp Biol Med. 1982;169:36-40.
40. Ma Y. et al. “Association Between Carbohydrate Intake and Serum Lipids.” J Am Coll Nutr. Apr 2006;25(2):155-163
41. Preuss, H. G. “Sugar-Induced Blood Pressure Elevations Over the Lifespan of Three Substrains of Wistar Rats.” J Am Coll of Nutr. 1998;17(1) 36-37.
42. Furth, A. and Harding, J. “Why Sugar Is Bad For You.” New Scientist. Sep 23, 1989;44.
43. Lee AT, Cerami A. “Role of Glycation in Aging.” Ann N Y Acad Sci. Nov 21, 1992 ;663:63-70.
44. Appleton, N. Lick the Sugar Habit.  (New York: Avery Penguin Putnam:1988).
45. Henriksen H. B. and, Kolset S.O.  Tidsskr Nor Laegeforen. Sep 6, 2007;127(17):2259-62.
46. Cleave, T.: The Saccharine Disease. (New Canaan Ct: Keats Publishing, Inc., 1974).131.
47. Ibid. 132.
48. Vaccaro O., et al. “Relationship of Postload Plasma Glucose to Mortality with 19 Year Follow-up.”  Diabetes Care. Oct 15,1992;10:328-334.
Tominaga, M., et al, “Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose.” Diabetes Care. 1999:2(6):920-924.
49. Lee, A. T. and Cerami, A. “Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging.” Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
50. Monnier, V. M. “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” J Gerontol. 1990:45(4 ):105-110.
51. Dyer, D. G., et al. “Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging.” J Clin Invest. 1993:93(6):421-422.
52. Veromann, S. et al. ”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica. Jul-Aug 2003 ;217(4):302-307.
53. Monnier, V. M. “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” J Gerontol. 1990:45(4):105-110.
54. Schmidt A.M. et al. “Activation of Receptor for Advanced Glycation End Products: a Mechanism for Chronic Vascular Dysfunction in Diabetic Vasculopathy and Atherosclerosis.” Circ Res. Mar 1999 19;84(5):489-97.
55. Lewis, G. F. and  Steiner, G. “Acute Effects of Insulin in the Control of VLDL Production in Humans. Implications for The Insulin-resistant State.” Diabetes Care. Apr 1996;19(4):390-3
R. Pamplona, M. .J., et al.  “Mechanisms of Glycation in Atherogenesis.” Med Hypotheses. 1990;40:174-181.
56. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.
57. Appleton, N. Lick the Sugar Habit. (New York: Avery Penguin Putnam, 1988).
58. Hellenbrand, W. ”Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study.” Neurology. Sep 1996;47(3):644-650
Cerami, A., et al. “Glucose and Aging.” Scientific American. May 1987: 90.
59. Goulart, F. S. “Are You Sugar Smart?” American Fitness.  Mar-Apr 1991: 34-38.
60. Scribner, K.B. et al. “Hepatic Steatosis and Increased Adiposity in Mice Consuming Rapidly vs. Slowly Absorbed Carbohydrate.” Obesity. 2007;15:2190-2199.
61. Yudkin, J., Kang, S. and Bruckdorfer, K. “Effects of High Dietary Sugar.”  Brit J Med.  Nov 22, 1980;1396.
62. Goulart, F. S. “Are You Sugar Smart?” American Fitness. March-April 1991: 34-38
63. Ibid.
64. Ibid.
65. Ibid.
66. Ibid.
67. Nash, J. “Health Contenders.” Essence. Jan 1992-23: 79-81.
68. Grand, E. “Food Allergies and Migraine.” Lancet. 1979:1:955-959.
69. Michaud, D. ”Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study.” J Natl Cancer Inst. Sep 4, 2002 ;94(17):1293-300.
70. Schauss, A.  Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981).
71. Peet, M. “International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological Analysis.” Brit J Psych. 2004;184:404-408.
72. Cornee, J., et al. “A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France,” Eur J Epidemiol. 1995;11:55-65.
73. Yudkin, J.  Sweet and Dangerous. (New York: Bantam Books,1974) 129.
74. . Choi HK, and Curhan G. “Soft drinks, fructose consumption, and the risk of gout in men: prospective cohort study.”  British Medical Journal.  Feb. 9,2008;336(7639):309-312.
75. Reiser, S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans.” Am J Clin Nutr. 1986:43;151-159.
76. Reiser,S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans.” Am J Clin Nutr. 1986;43:151-159.
77. Molteni, R, et al. “A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning.” NeuroScience. 2002;112(4):803-814.
78. Monnier, V., “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” J Gerontol. 1990;45:105-111.
79. Frey, J. “Is There Sugar in the Alzheimer’s Disease?” Annales De Biologie Clinique. 2001; 59 (3):253-257.
80. Yudkin, J. “Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes.” Nutrition and Health. 1987;5(1-2):5-8.
81. Ibid.
82. Blacklock, N. J., “Sucrose and Idiopathic Renal Stone.”  Nutrition and Health. 1987;5(1-2):9-12.
Curhan, G., et al. “Beverage Use and Risk for Kidney Stones in Women.” Ann Inter Med. 1998:28:534-340.
83. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.
84.  Moerman, C. J., et al. “Dietary Sugar Intake in the Etiology of Biliary Tract Cancer.” Internat J Epidemiol. Apr 1993;2(2):207-214.
85.  Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents.” Am J Nutr. Jun 1997;1113-1117.
86.  Ibid.
87. Bostick, R. M., et al. “Sugar, Meat.and Fat Intake and Non-dietary Risk Factors for Colon Cancer Incidence in Iowa Women.” Cancer Causes & Control. 1994:5:38-53.
88.  Ibid.
Kruis, W., et al. “Effects of Diets Low and High in Refined Sugars on Gut Transit, Bile Acid Metabolism and Bacterial Fermentation.” Gut. 1991;32:367-370.
Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating, And Obesity.” Pediatrics. Mar 1999;103(3):26-32.
89.  Yudkin, J and Eisa, O. “Dietary Sucrose and Oestradiol Concentration in Young Men.” Ann Nutr Metabol. 1988:32(2):53-55.
90.  Lee, A. T. and Cerami A. “The Role of Glycation in Aging.” Ann N Y Acad Sci. 1992; 663:63-70.
91.  Moerman, C. et al.”Dietary Sugar Intake in the Etiology of Gallbladder Tract Cancer.” Internat J of Epi. Apr 1993; 22(2):207-214.
92. Avena N.M. “Evidence for Sugar Addiction: Behavioral and Nuerochemical Effects of Intermittent, Excessive Sugar Intake.” Neurosci Biobehav Rev. 2008;32(1):20-39.
Colantuoni, C., et al. “Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence.” Obes Res. Jun 2002 ;10(6):478-488.
93. Ibid.
94. The Edell Health Letter. Sept 1991;7:1.
95. Sunehag, A. L., et al. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition.” Diabetes. 1999 ;48 7991-8000).
96. Christensen L. et al. “Impact of A Dietary Change on Emotional Distress.” J Abnor Psychol. 1985;94(4):565-79.
97. Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating and Obesity.” Pediatrics. Mar1999;103(3):26-32.
98. Girardi, N.L.” Blunted Catecholamine Responses after Glucose Ingestion in Children with Attention Deficit Disorder.” Pediatrics Res. 1995;38:539-542.
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99. Blacklock, N. J. “Sucrose and Idiopathic Renal Stone.” Nutrition and Health. 1987;5(1 & 2):9-17.
100. Lechin, F., et al. “Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans.” Neurophychobiology. 1992;26(1-2):4-11.
101. Arieff, A. I.  “IVs of Sugar Water Can Cut Off Oxygen to the Brain.” Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86.
102. De Stefani, E. “Dietary Sugar and Lung Cancer: a Case Control Study in Uruguay.” Nutr and Cancer. 1998;31(2):132_7.
103. Sandler, B.P.  Diet Prevents Polio.  (Milwakuee, WI,: The Lee Foundation for Nutritional Research, 1951).
104. Murphy, P. “The Role of Sugar in Epileptic Seizures.” Townsend Letter for Doctors and Patients. May, 2001.
105. Stern, N. & Tuck, M. “Pathogenesis of Hypertension in Diabetes Mellitus.” Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (Phil. A: Lippincott Williams & Wilkins, 2000)943-957.
106. Christansen, D. “Critical Care: Sugar Limit Saves Lives.” Science News. June 30, 2001;159:404.
107. Donnini, D. et al. “Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.” Biochem Biohhys Res Commun.  Feb 15, 1996:219(2):412-417.
108. Levine, A.S, et al. “Sugars and Fats: The Neurobiology of Preference “ Am J Nutr. 2003 133:831S-834S.
109. Schoenthaler, S. “The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six Institutional Settings.” Int J Biosocial Res. 5(2):88-89.
110. Deneo-Pellegrini H,. et al.Foods, “Nutrients and Prostate cancer: a Case-control study in Uruguay.” Br J Cancer. 1999 May;80(3-4):591-7.
111. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition.” Diabetes. 1999 Apr;48(4):791-800.
112. Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake Among Pregnant Adolescents.” Am Jf Nutr. 1998;128:807-1810.
113. Peet, M. “International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological Analysis.” Brit J Psychiatry. 2004;184:404-408.
114. Fonseca, V. et al.  “Effects of a High-fat-sucrose Diet on Enzymes in Homosysteine Metabolism in the Rat.” Metabolism. 200; 49:736-41.
115. Potischman, N, et.al.  “Increased Risk of Early-stage Breast Cancer Related to Consumption of Sweet Foods among Women Less than Age 45 in the United States.” Cancer Causes Control. 2002 Dec;13(10):937-46.
116. Negri. E. et al. “Risk Factors for Adenocarcinoma of the Small Intestine.”
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128. Shaw, Gary M. et al. “Neural Tube Defects Associated with Maternal Periconceptional Dietary Intake of Simple Sugars and Glycemic Index.”
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What Your pH Says About You–Alkaline/Alkaline

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pH Paper

Urine/Saliva pH Test Paper


With the urine and saliva both above 6.5 it shows the urine, saliva, & average to be alkaline.  This arrangement will mean that the person is in a biochemical pattern for constipation. The system is overloaded with toxins from the usual metabolic wastes of cell function and also from the bowel reabsorbing putrefactive chemicals due to the fecal matter remaining in the system too long.  The presence of parasites is very likely in this range, since free room and board is being provided. This pH configuration will aggravate deterioration of disks, especially those in the back.  Deterioration of cartilage tissue is also aggravated.  Upper respiratory and sinus congestion will be aggravated, as will lymph congestion.   This type of person may be prone to lung infections.  In the years when tuberculosis was  a problem, this type of chemistry would have made this person very likely to come down with the disease.  Body odors, from breath to feet to bowel gas, will be symptoms related to this pattern.  Anatomically, this type of person would have an abdomen that would become more distended with age (other reasons also play into this).  In later life, there is a tendency to be overweight.  There is often a problem with skin pigmentation, because this pattern usually means that vitamin D is ratioed in the direction of excess.  An anionic pH will increase the production of melanin, or skin pigment, causing more energy to be absorbed from the sun, resulting in greater vitamin D production.  Blood pressure can be affected. Various types of skin tumors and lesions, boils and cysts may develop.  Tooth decay can be excessive, especially when the overall diet has been poor.  Women may complain of cracking, splitting, and ridged fingernails.  Women’s menstrual cycles can become very uncomfortable, with excessive hemorrhage and/or cramping, because of the body trying to use the uterus as a point of dumping toxins from other areas of the body.  PMS is a potential in this configuration.  The right side of the body will be the weak side, which means that this person will have a strong tendency for developing curvature of the spine to the left, as the body pulls toward the strong side.  Of course, this depends on how long this pattern has been in existence.  You may not realize it, but a person with this anionic pH pattern will have a tendency to cross the left leg over the right when sitting or favor standing on the left leg (strong side) while standing for long periods of time.  From a chiropractic standpoint, the left leg will tend to be shorter than the right, unless the person prefers sleeping on the strong side (left).  In that situation the spine will have an “S” curve configuration, meaning that the right hip will be higher than the usual strong side.  Vitamin C is very lacking in the system; and, in this situation, high levels of vitamin C can be used to provide extra cations in the diet to help lower the pH.  Mega doses of vitamin C may be used in this pattern, depending on how strongly anionic the pH, but, remember,  when the body is forced to change pH ranges without the understanding of how mineral energy, especially calciums, fit into the picture, the body will not respond consistently.  The excess vitamin C may result in aggravating excessive gas and/or diarrhea.

Excerpted from Biological Ionization as it Applies to Human Nutrition by A. F. Beddoe

To Your Health,

Coach Debbie

RBTI: A Few Rules by Brixman

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Don’t Buy Into BG12, It’s a Chemical Fake

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BG12 Approved by the FDA!

BG12 is basically a chemical Knockoff of Protandim.  In a clinical study doing a comparison between “The Real Thing” and the Knockoff, Protandim won hands down.

So, if Protandim won, why would the pharmaceuticals keep attempting to get their drug approved?  Money of course. Protandim retails at $50 a bottle. 1 bottle lasts 1 person 1 month. At retail, Protandim will only cost $600.00 a year for someone with Multiple Sclerosis.  The BG12 that just got approved by the FDA for use with Multiple Sclerosis is projected to cost $50,000.00 a year, the side effects are free! 

BG-12, tBHQ, SFN and protandim are well-tolerated and strongly induce Nrf2-driven antioxidant enzyme production in oligodendrocytes, with protandim showing the most potent induction.

See the original document here: http://registration.akm.ch/einsicht.php?XNABSTRACT_ID=137548&XNSPRACHE_ID=2&XNKONGRESS_ID=150&XNMASKEN_ID=900

With Protandim, there are no side effects, no known drug interactions and no real limits to the possibilities of what it could do for you.  The University of Colorado performed a human clinical trial published in 2006 that proved Protandim works in 100% of people to reduce their oxidative stress markers by an average of 40% in just 30 days. That wasn’t just some people, it was ALL people.

If MS is an issue for you, perhaps you should consider Protandim. Take the Amsterdam study to your Doctor and ask him why he would rather use a chemical when he could use a clinically proven herbal supplement that has no negative effects. He might just try it.

Take a natural approach to overcoming your MS symptoms. Sign up for a Health Evaluation with me, Coach Debbie, and lets see if Protandim may be right for you.  I offer a 6-month Healthier Habits coaching program to help you succeed in overcoming your obstacles. It all starts with a FREE Health Evaluation. Sign up today!


Protandim is a supplement. Protandim, LifeVantage, and Coach Debbie do not claim to diagnose, treat, cure or mitigate any diseases. If you have a medical complaint, please consult with your MD.

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